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. 2021 Nov 22;25(1):24. doi: 10.3892/mmr.2021.12540

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Copyright: © Gao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Sch A relieves fibrosis of neonatal rat ventricular myocytes via miR-155. (A) The transfection efficiency of miR-155 mimic or miR-155 inhibitor was evaluated by RT-qPCR. (B) The expression of miR-155 was determined via RT-qPCR. U6 was used as a control. (C and D) The level of α-SMA was observed by immunofluorescence. Scale bar, 50 µm; magnification, ×200. ^&&&P<0.001 vs. MC; ^ΔΔΔP<0.001 vs. IC; ***P<0.001 vs. Blank; ^▲▲▲P<0.001 vs. ISO + IC; ^###P<0.001 vs. ISO; ^{^^^}P<0.001 vs. ISO + Sch A + MC. Sch A, Schisandrin A; miR, microRNA; RT-qPCR, reverse transcription-quantitative PCR; α-SMA, α-smooth muscle actin; M, miR-155 mimic; MC, miR-155 mimic control; I, miR-155 inhibitor; IC, miR-155 inhibitor control; ISO, isoproterenol.