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TNEA treatment promoted the degradation of beta-amyloid precursor protein (APP) fragments in 5XFAD mice brains. Representative western blots showed the levels of full-length APP (Fl-APP), carboxy-terminal fragments (CTFs) and BACE1/β-secretase 1 in the prefrontal cortex (PFC) (A) and hippocampus (HI) (B) of mice brains. Data were quantified as mean ± SEM (male, n = 6). *p < 0.05, **p < 0.01 vs. 5XFAD group analyzed by Unpaired t test
