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. 2021 Dec 1;12(12):1123. doi: 10.1038/s41419-021-04403-2

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — A Mice were intraperitoneally injected with GS9973 (20 mg/kg, q2d), 4 weeks after CCL4 injection, the degree of liver fibrosis was observed by photographs, HE, masson, Sirius red staining, and immunohistochemistry of α-SMA; B Mice were injected with GS9973 (20 mg/kg, q3d), the protein level of TGF-β in the liver was measured after 8 weeks of CCL4 injection; C The mRNA level of α-SMA was evaluated after 4 weeks; D The administration of GS9973 (20 mg/kg, q2d, ip) caused the death of mice with BDL.