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. 2021 Dec 1;12(12):1120. doi: 10.1038/s41419-021-04409-w

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — A Comparison of pulmonary metastases in mice (n = 3). B Survival analysis showed that exosome miR-18b reduced survival rate in mice. C qRT-PCR detection of miR-18b expression in metastatic cells (n = 3). D Western blot indicated the expression of TCEAL7, E-cadherin, and Vimentin in mouse lung metastatic cells (n = 3). E A proposed model illustrating the role of CAF-derived exosomal miR-18b promotes BC cells invasion and metastasis by downregulation of TCEAL7. * indicates p < 0.05. The measurement data were expressed using mean ± SD, and the experiment was repeated three times. Comparisons between two groups are analyzed by t-test.