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. 2021 Dec 4;20:158. doi: 10.1186/s12943-021-01457-w

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — The inhibitory effect of circAXIN1 siRNA is rescued by linear AXIN1-295aa. a Cell proliferation of AGS and N87 is inhibited by circAXIN1 siRNA and reversed by linear AXIN1-295aa transfection. b Cell migration of AGS (treated with 10 μg/mL mitomycin for 1 h) and invasion of AGS and N87 are both inhibited by circAXIN1 siRNA and rescued with linear AXIN1-295aa transfection. c The inhibitory effect of circAXIN1 siRNA on cell migration is reversed by linear AXIN1-295aa. AGS cells are treated with 10 μg/mL mitomycin for 1 h in the wound healing assay, 48 h after transfection. d Colony formation ability is suppressed in AGS cells by circAXIN1 siRNA and rescued by linear AXIN1-295aa transfection