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. 2021 Nov 24;49(11):03000605211058376. doi: 10.1177/03000605211058376

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© The Author(s) 2021

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 1. — Chemerin/CMKLR1 was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. *P < 0.05, **P < 0.01, ***P < 0.001 compared with control group.