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. 2021 Dec 1;14:6397–6407. doi: 10.2147/JIR.S339133

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© 2021 Sun et al.

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The overexpression of circFoxo3 attenuates MI-induced cardiac dysfunction in rat model. (A–H) The MI Rat model was established and treated with injection of circFoxo3 vectors or control vectors surrounding the infarcted region. (A) Relative level of circFoxo3 RNA in heart tissues was detected by qRT-PCR. (B) HE staining of inflammatory cell infiltration in heat tissues three days after surgery. (C–G) Infarcted size,²⁵ left ventricular fractional shortening (LVFS) (D), Left ventricular ejection fraction (LVEF) (E), left ventricle anterior wall thickness in diastole (LVAWd) (F), and left ventricular posterior wall end diastole (LVPWd) (G) were measured. (H) The apoptosis was detected by TUNEL staining. **p < 0.01.