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. 2021 Sep 12;78(6):802–808. doi: 10.1097/FJC.0000000000001123

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Copyright © 2021 The Author(s). Published by Wolters Kluwer Health, Inc.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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FIGURE 4. — Overexpression of MFACR increased AC16 cell apoptosis induced by hypoxia through miR-125b. Under hypoxic conditions, the role of MFACR and miR-125b in regulating the apoptosis of AC16 cells was analyzed by cell apoptosis assay. *P < 0.05.