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. 2021 Dec 6;11:23443. doi: 10.1038/s41598-021-02761-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Profiles of genetic alterations in 90 gastric cancers. Genetic alterations of 46 cancer-related genes were analyzed by next-generation target sequencing. Among the 90 gastric cancers, 47 cancers had 68 somatic point mutations of 15 different genes, such as TP53, KRAS, and PIK3CA. Ten cancers had gene amplification of one of ERBB2, KRAS, PIK3CA, and MET. Gastric cancers in individual groups and genes analyzed were aligned in the order of the number of total mutations and mutation frequency, respectively. Black and red boxes show somatic point mutations and gene amplifications, respectively. Gastric cancers with current and past infection had comparable profiles of somatic point mutations and gene amplifications.