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. 2021 Dec 6;11:23443. doi: 10.1038/s41598-021-02761-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Frequency of point mutations and gene amplification in gastric cancers with current and past H. pylori infection. (a) Frequency of point mutations. Mutation frequencies of TP53, KRAS, and PIK3CA were similar between gastric cancers with current H. pylori infection (TP53, 38%; KRAS, 2%; and PIK3CA, 4%) and those with past infection (TP53, 31%; KRAS, 9%; and PIK3CA, 6%). Black and white bars show frequencies in gastric cancers with current and past H. pylori infection, respectively. (b) Frequency of gene amplification of ERBB2, KRAS, PIK3CA, and MET. The frequency was similar between gastric cancers with current H. pylori infection (ERBB2, 9%; and KRAS, 2%) and those with past infection (ERBB2, 3%; and KRAS, 3%). Black and white bars show frequencies in gastric cancers with current and past H. pylori infection, respectively.