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. 2021 Dec 8;12(12):1140. doi: 10.1038/s41419-021-04427-8

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — A The correlation coefficient between risk score and immune checkpoints. B The distribution of PD-L1 protein level in the high- and low-risk groups based on the TCPA datasets. **, p < 0.01. C The distribution of TIM3 protein level in the high- and low-risk groups by IHC staining. Scale bar, 50 μm. D The distribution of PD-1 expression in GBM patients before treatment with PD-1 inhibitors. **, p < 0.01. E The TIDE score and response results to immunotherapy of patients with glioma. F Fraction of PTEN loss in the low-risk group versus high-risk group. G GSEA enrichment score of gene-set KIM_PTEN_TARGETS_UP for the low-risk group versus high-risk group. ****, p < 0.0001. H Heatmap showing the gene sets differentially enriched in the low-risk group versus high-risk group.