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The natural history of endometriosis. In susceptible women, additional G-E incidents (driven by oxidative stress from retrograde menstruation and infection) induce clonal and heterogenous hetrogeneous (aromatase and progesterone resistance) lesions, which develop in the peritoneal cavity which is a specific microenvironment. Inhibition of growth by progressive fibrosis (because of immunology and bleeding in the lesions) results in variable severity of lesions.
