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. 2021 Jun 23;57(1):95–105. doi: 10.1097/SHK.0000000000001823

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Copyright © 2021 by the Shock Society

This article is made available via the PMC Open Access Subset for unrestricted re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the COVID-19 pandemic or until permissions are revoked in writing. Upon expiration of these permissions, PMC is granted a perpetual license to make this article available via PMC and Europe PMC, consistent with existing copyright protections.

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Fig. 4 — Comparison of circulating endothelial biomarkers between critical COVID-19 and septic shock patients. Datapoints indicate individual measurements, whereas horizontal bars show median and interquartile ranges. C5b9 deposits are expressed as fold increase versus control. ADAMTS-13 indicates a disintegrin-like and metalloprotease with thrombospondin type 1 motif no. 13; NETs, neutrophil extracellular traps; PAI-1, plasminogen activator inhibitor-1; sC5b9: soluble C5b9; sTNF-RI, soluble tumor necrosis factor receptor I; sVCAM-1, soluble vascular cell adhesion molecule-1; VWF, Von Willebrand Factor.