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. 2021 Nov 26;12:774539. doi: 10.3389/fphar.2021.774539

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Copyright © 2021 Dai, Liu, Peng, Qu, Lin, Peng and Li.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Entinostat suppresses OGD-induced neuronal injury. In the OGD-induced neuronal damage model, cell viability was measured after being treated with 0, 1, 2, or 5 μM entinostat for 24 h (A) or treated with 2 μM entinostat for 12, 24, 36, or 48 h (B). (C) PI staining of primal neurons at 24 h after being treated with 2 μM entinostat, and DAPI stained all cell nuclei. (D) Histogram analysis of PI-positive cells. Scale bars: 50 μm. Data are expressed as the mean ± SEM, n = 4. *p < 0.05, vs. the control group; #p < 0.05, vs. the OGD group.