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. 2021 Dec 7;15(1):101302. doi: 10.1016/j.tranon.2021.101302

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© 2021 The Authors. Published by Elsevier Inc.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 1 — Inhibitory effect of nafamostat mesylate on cell viability of endocrine-resistant estrogen receptor-positive breast cancer. (A) IC₅₀ values of H184, parental MCF7, MCF7-TamR, and MCF7-FulR cells for NM. (B) Analysis of the viability of H184, parental MCF7, MCF7-TamR and -FulR cells treated with various doses of NM for 72 and 96 h, using MTT assays. (C) Analysis of growth of MCF7-TamR (upper) and MCF7-FulR (lower) cells, assessed using colony formation assays. (D) Quantitative analysis of colony formation assays for NM-treated MCF7-TamR (left) and MCF7-FulR (right) cells. Data are represented as the mean ± SEM for biological triplicate experiments. ^⁎#P < 0.01, compared with the results for untreated H184, parental MCF7, MCF7-FulR, and MCF7-TamR cells.