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. 2021 Nov 29;12:683813. doi: 10.3389/fpls.2021.683813

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Copyright © 2021 Li, Dong, Jia, Huang, Zou, An, Song and Chen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Hypothetical view of GmMTP8.1 affecting Mn tolerance in transgenic Arabidopsis. Endoplasmic reticulum (ER)-localized GmMTP8.1 is involved in Mn transport into the ER, which has probably contributed in reduction of cytosolic Mn and maintenance of Mn homeostasis in transgenic Arabidopsis overexpressing GmMTP8.1. Furthermore, cytosolic Mn in leaf can be transported into the Golgi apparatus by the involvement of Golgi-localized AtMTP11 and ECA3, and then sequestered into vesicles and removed from the cell via exocytosis, thereby decreasing cellular Mn levels in shoot. The upregulation of AtNRAMP3 might also help to maintain Mn homeostasis in leaves of transgenic Arabidopsis. In addition, GmMTP8.1 has likely mediated Mn tolerance, along with AtMTP8, by increasing Mn sequestration into root vacuoles, conferring Mn tolerance. White and black arrows represent import into and export out of the cytosol, respectively.