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. 2021 Dec 14;19:508. doi: 10.1186/s12967-021-03173-x

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — GLUT1 inhibitor (BAY-876) was effective to inhibit the progression of TRIM38^low/− bladder cancer. a The half maximal inhibitory concentration (IC50) values of BAY-876 (GLUT1 inhibitor) in BLCA cell lines (T24, EJ and 5637). b–d BAY-876 could suppress the cell growth of BLCA cells in a dose-dependent manner. e Representative image inhibited that BAY-876 could inhibit the tumor volumes derived from TRIM38-KO cells relative to tumors derived from control cells. f The tumor volumes were detected and recorded at the indicated timepoints and the growth curve was generated. g Tumors were resected and weighed to compare the differences in the indicated groups