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. 2021 Dec 15;73(Suppl 5):S442–S453. doi: 10.1093/cid/ciab885

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© The Author(s) 2021. Published by Oxford University Press for the Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 4. — Pulmonary histological findings of 80 fatal cases of coronavirus disease 2019 (COVID-19), ultrasound-guided minimally invasive tissue sampling. All scale bars = 100 µm unless otherwise specified. A, Severe proliferative/organizing diffuse alveolar damage with lymphocytic septal infiltrate, and fibroblast intra-alveolar proliferation, depositing collagen matrix. Hematoxylin and eosin (H&E) staining. B, Collagen matrix deposition in alveolar spaces. Masson trichrome staining. C, Severe interstitial fibrosis with “pseudo-cystic” formations due to barotrauma. H&E staining. D, Septal capillaries with angiomatoid aspect associated with tiny fibrin clots. H&E staining. E, Thrombus in a pulmonary artery, with surrounding parenchyma exhibiting coagulative necrosis. H&E staining. F, Secondary suppurative bacterial pneumonia. H&E staining. G, Grocott silver stain showing hyphae compatible with Candida spp in a case of COVID-19 with bacterial pneumonia. Scale bar = 20 µm. H, Pulmonary miliary tuberculosis associated with COVID-19. H&E staining.