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. 2021 Dec 15;13:200. doi: 10.1186/s13195-021-00943-z

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Main effects of Aβ on biomarkers and cognitive measures in non-dementia participants. The figure shows the associations of baseline CSF Aβ42 on longitudinal biomarkers and cognitive measurements in CN and MCI populations, respectively. p values were extracted from linear mixed-effects models adjusted for age, sex, APOE ε4 carrying status, and educational levels. CN, normal controls; MCI mild cognitive impairment; Aβ, amyloid-β; p-tau, phosphorylated tau; t-tau, total tau; NFL, neurofilament light; sTREM2, soluble triggering receptor on myeloid cells 2; MEM, memory function; EF, executive function; LAN, language; VS, visuospatial functioning