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. 2021 Dec 6;49(22):13045–13061. doi: 10.1093/nar/gkab1148

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© The Author(s) 2021. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (https://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 6. — HDNMT2 recognizes well-folded tRNA substrates. (A) The capacity of a tRNA^Gly(GCC) mutant lacking the CCA terminus to be methylated by hDNMT2. (B) Schematic diagram showing the truncations of the D-loop and T-loop of tRNA^Gly(GCC), respectively. The capacity of the two truncated tRNA^Gly(GCC) to be methylated by hDNMT2. (C) Schematic diagram showing the tRNA^Gly(GCC)-minihelix, which is formed by the entire anticodon stem loop fused with the acceptor stem. The capacity of the tRNA^Gly(GCC)-minihelix to be methylated by hDNMT2.