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Model for CAMSAP3 regulating NSCLC senescence‐associated phenotypes. In the presence of CAMSAP3 (left), CAMSAP3 binds to vimentin and maintains vimentin‐p‐ERK interaction, consequently stabilizing ERK activation. An active ERK promotes cyclin D1 expression and induces normal cell proliferation. In the absence of CAMSAP3 (right), the interaction of vimentin and p‐ERK is reduced. Thus, senescence‐associated phenotypes are activated
