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. 2021 Dec 20;7:397. doi: 10.1038/s41420-021-00797-2

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Young and aged mice were administered TAA (200 mg/kg) and PBS as a control. A Serum ALT (left panel) and AST (right panel) levels in each group. B Representative H&E staining of the liver. C Representative TUNEL (green fluorescence) staining of the liver with DAPI counterstaining (blue fluorescence) and D quantification. E Immunoblotting of Bax and Bcl-2 expression in liver tissue from each group and F quantification. All data shown represent n = 8–10 mice per group. All results are representative of at least three independent experiments. Values are presented as the mean ± SD. *p ≤ 0.05; **p ≤ 0.01; ***p ≤ 0.001.