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Schematic representation of the potential mechanism by which EGCG attenuates pressure overload-induced myocardial remodeling. EGCG may ameliorate myocardial remodeling by regulating the overexpression of HDAC5. ANP, atrial natriuretic peptide; BNP, brain natriuretic peptide; EGCG, epigallocatechin gallate; H3K27ac, histone acetylated lysine 27 on histone H3; HDAC5, histone deacetylase 5; MEF2A, myocyte enhancer factor 2A.
