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. 2021 Dec 2;6(1):bvab169. doi: 10.1210/jendso/bvab169

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© The Author(s) 2021. Published by Oxford University Press on behalf of the Endocrine Society.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — Δ Glycated hemoglobin A_1c (HbA_1c) following the curative procedure for Cushing syndrome. A, Δ HbA_1c in all patients (n = 130). B, Δ HbA_1c in 42 patients in whom hyperglycemia management was not changed (including those on no medications). C, Δ HbA_1c in 71 patients in whom the intensity of hyperglycemia management was decreased (decrease in dose or number of medications). D, Δ HbA_1c in 17 patients in whom the intensity of hyperglycemia management was increased (increase in dose or number of medications, new start of medications).