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Diagram of DUXAP8’s role in HCC and its mechanism. Mettl3-mediated m6A methylation modification stabilizes and upregulates DUXAP8, which competitively binds to miR-584-5p through a ceRNA mechanism, thereby targeting MAPK1 and activating the MAPK/ERK pathway to regulate the malignant phenotype and chemotherapy resistance of HCC.
