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. 2021 Dec 20;10(12):2020. doi: 10.3390/antiox10122020

Figure 7.

Figure 7

Silencing of CCN2 alleviated oxidative stress response induced by AR-injury in cultured PTECs. (A) Representative Western blotting of protein extracts from cultured cells for 4-HNE, nitrotyrosine, and p-p53 showed that increased oxidative stress response and DDR in AR-injured PTECs was alleviated by silencing of CCN2. Data represent three independent experiments. (B) Malondialdehyde (MDA) levels indicated that increased oxidative stress response in AR-injured PTECs was alleviated by silencing of CCN2. Data represent six independent experiments. Data are expressed as the mean ± SEM. *** p < 0.001.