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Schematic representation of the interactions of (endo)cannabinoids and synthetic cannabinoid compounds with microglia in the context of epilepsy and neuroinflammation. Endocannabinoids (green) interact with CB2 receptors and other non-classical receptors (GPR55, PPAR and abn-CBDR). Fatty acid amides, such as PEA, interact with non-classical receptors (yellow), while synthetic cannabinoid effects (brown) involve mainly CB1 and CB2 receptors.
