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. Author manuscript; available in PMC: 2023 Jan 1.
Published in final edited form as: Prog Neurobiol. 2021 Oct 17;208:102181. doi: 10.1016/j.pneurobio.2021.102181

Figure 2 |. Age-dependent analysis of transposable element RNA levels in three different mouse models of tauopathy.

Figure 2 |

a. The rTg4510 mouse model of tauopathy. b. Differentially expressed transposable elements in the female rTg4510 mouse cortex from three to nine months based on RNA-seq. Differentially expressed transposable elements were grouped into families and represented on the graph. n=5–6 biological replicates per age. c. The proportion of different types of differentially expressed transposable element subfamilies within each transposable element family. d. The JNPL3 mouse model of tauopathy. e. Differentially expressed transposable elements in the female JNPL3 spinal cord from two to twelve months based on RNA-seq. n=3–6 biological replicates per age. f. The proportion of differentially expressed transposable element subfamilies within each transposable element family. g. The PS19 mouse model of tauopathy. h. Differentially expressed transposable elements in the male PS19 spinal cord at four and nine months based on RNA-seq. Differentially expressed transposable elements were grouped into families and represented on the graph. n=5–7 biological replicates per age. I. The proportion of differentially expressed transposable element subfamilies within each transposable element family. An adjusted P value of <0.05 was considered significant.