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. 2021 Dec 14;12:727371. doi: 10.3389/fendo.2021.727371

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Copyright © 2021 Cao, Chen, Zhang, Wu, Zeng, Zhang and Cai

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Chronic effects of CLZ on glucose metabolism. (A) Biochemical parameters including fasting plasma glucose (H = 28.96, p < 0.0001), HbA1c (H = 28.21, p < 0.0001), insulin (H = 21.25, p = 0.0003), hepatic glycogen content (H = 25.56, p < 0.0001) and glucagon levels (H = 16.08, p = 0.0029). (B) H&E staining. (C) Representative images of PAS staining of liver sections, showing that the decreased glycogen in CLZ-treated rat was prevented by PGRMC1-OE. Red: glycogen; purple: nuclei of liver cells.