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. 2021 Dec 14;12:775680. doi: 10.3389/fphar.2021.775680

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Copyright © 2021 Dai, Cai, Chen, Wang, Zhang, Wang, Tu, Zhu, Li and Lu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Bru induced apoptotic cell death of glioblastoma cells. (A) The cell apoptosis of A172 cells, U251 cells, and U87 increased obviously after treatment with Bru by flow cytometry analysis. The results revealed that Bru indeed rendered A172 cells, U251 cells, and U87 to undergo apoptosis. (B) A172, U251, and U87 cells were treated with Bru as indicated, and apoptosis-associated proteins were analyzed by Western blot using antibodies against Bax, Bcl-2, pro-Caspase-3, cleaved-Caspase-3, pro-Caspase-9, and cleaved-Caspase-9. Beta-Actin was used to control the consistency of protein loading.