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. 2021 Dec 30;21:302. doi: 10.1186/s12906-021-03479-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Effect of SE treatment on inflammation, fibrosis and apoptosis induced by CCl₄ in liver. The relative gene expression of; (I) NF-KB, (II) TNF-α, (III) IL-6, (IV) TGF-β, (V) p53. C group: oral administration with water; CCl₄ group: rats injected with CCl₄. SE group: rats received SE only. SE-CCl₄ group: rats received SE before and during CCl₄ administration. SE-CCl₄-SE group: rats received SE before, during and after CCl₄ administration. The results are shown as mean ± SD (n = 8). Different letters for the same parameter are significantly different at p < 0.05