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. 2021 Apr 15;35:279–294. doi: 10.1016/j.jare.2021.04.003

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© 2021 The Authors. Published by Elsevier B.V. on behalf of Cairo University.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 2 — Diagrams showing vitamin C relieves FOA-induced hepatotoxicity by controlling gene expression. The comparative transcriptomic analysis reveals differential gene expression induced by vitamin C (PFOA vs vitamin C + PFOA group). (a) The DEGs were assessed by the Gene Ontology (GO) enrichment analysis. The rich factor plot shows that the vitamin C-relieved PFOA-altered biological processes (p < 0.05). (b) Data from gene network analysis using IPA, demonstrating cell signaling involved in the protective action of vitamin C.

Fig. 2 — Diagrams showing vitamin C relieves FOA-induced hepatotoxicity by controlling gene expression. The comparative transcriptomic analysis reveals differential gene expression induced by vitamin C (PFOA vs vitamin C + PFOA group). (a) The DEGs were assessed by the Gene Ontology (GO) enrichment analysis. The rich factor plot shows that the vitamin C-relieved PFOA-altered biological processes (p < 0.05). (b) Data from gene network analysis using IPA, demonstrating cell signaling involved in the protective action of vitamin C.