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. 2021 Nov 13;44(1):29–38. doi: 10.1007/s13258-021-01152-6

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Schematic diagram of the proposed molecular mechanisms. A schematic diagram depicts molecular basis of the inhibition of FOSL2 aggravates the apoptosis of ovarian cancer cells through promoting the formation of inflammasome. Inhibition of FOSL2 could activate the inflammasome via up-regulation the expression of ASC, caspase-1, IL-1β and IL-18 to promote the apoptosis and suppress the proliferation, migration and invasion of ovarian cancer cells