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. 2021 Oct 25;35(1):360–378. doi: 10.1007/s13577-021-00636-7

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Expression levels of USP30-AS1 and its promoter methylation sites in AML. A1 Global expression of lncRNA USP30-AS1 across 31 cancer types. A2 Expression levels of USP30-AS1 between the normal and LAML tumor tissues. *p < 0.05, Tumor tissues: n = 173; Normal tissues: n = 70. B Overall survival analysis between AML patients with low and high expression of USP30-AS1. C Effects of low- and high-methylation status across diverse methylation sites of USP30-AS1 promoter on survival in AML patients. *p < 0.05, n = 60. D Correlation analysis of expression of USP30-AS1 and different methylation status of promoter