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. 2021 Oct 25;35(1):360–378. doi: 10.1007/s13577-021-00636-7

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — USP30-AS1 regulates USP30 and ANKRD13A in cis in AML cells. A Schematic illustration of the USP30-AS1 and USP30/ANKRD13A locus, their promoters enriched in the H3K4me1, H3K4me3 and H3K27Ac proteins. B FISH analysis of the location of USP30-AS1 (red) in the nuclear of AML cells. C ChIP was performed to assess the enrichments of USP30 and ANKRD13A promoters in H3K4me1, H3K4me3 and H3K27Ac proteins. Significant differences are indicated by *p < 0.05, **p < 0.01 and ***p < 0.001, n = 3