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. 2021 Dec 3;53(12):1866–1876. doi: 10.1038/s12276-021-00704-w

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a t-SNE plot of the EC subpopulations from control and hypertensive tissue (control, n = 624 ECs; hypertension, n = 1056 ECs. EC 1, n = 1107 cells; EC 2, n = 302 cells; EC 3, n = 271 cells). b Expression levels of marker genes associated with 3 EC subpopulations across the 19 clusters. c Dot plot of the top 20 genes with specific expression for each EC subpopulation. d Module scores of 11 features (or functions) in control and hypertensive aortic EC subpopulations. e Bar graph of the composition of each EC subpopulation (dashed black line, expected proportion of cells in the control group). f Cell cycle stage distribution of 7 subpopulations from control and hypertensive tissue.