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. 2022 Jan 9;22:10. doi: 10.1186/s12906-021-03494-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Effects of acacetin on the survival and invasion of MKN45 and MGC803 cells. A The chemical structure of acacetin. B MKN45 and MGC803 cells were treated with 0–100 μM acacetin for 24–72 h and subjected to CCK-8 assays. C A wound healing assay was performed to evaluate the antimetastatic effect of acacetin in MKN45 and MGC803 cells (magnification, 10×). D Transwell migration and Matrigel invasion assays of MKN45 and MGC803 cells treated with or without acacetin were performed (magnification, 20×)