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. 2022 Jan 9;22:10. doi: 10.1186/s12906-021-03494-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Mechanism of acacetin in peritoneal metastasis of GC in nude mice. A Tumours were implanted in the abdomen, and then, the gross and microscopic pathological features of metastatic nodules in the livers of mice were observed. B The number of cancerous nodules colonizing the abdomen. C Changes in body weight in nude mice. D Difference in EMT-related protein expression in liver metastatic nodules of mice treated with different concentrations of acacetin. Data are expressed as the mean ± SD (n = 3). * p < 0.05, ** p < 0.01, *** p < 0.001 compared to the control group