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Gastrointestinal tract barrier function. (a) α1-antitrypsin in feces was significantly more in the KCASP1Tg mice than in the WT littermates (n = 6 per group). (b) However, intestinal permeability was not considered to be enhanced after evaluating the plasma concentration of FITC-dextran in the KCASP1Tg mice (n = 6 per group). All data were expressed as means ± SDs. ** p < 0.01 between KCASP1Tg and wild mice by Mann–Whitney test.
