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. 2022 Jan 11;13(1):51. doi: 10.1038/s41419-022-04500-w

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — miR-145-5p counteracted with linc01133 in the proliferation and cell cycle progression of gastric cnacer cells demonstrated by EdU (A, B), FASC (C) and MTS assay (D). Linc01133 with miR-145-5p binding sites mutation failed to accelerate the tumor cell growth demonstrated by EdU (E, F) and MTS (G). Each experiment was performed in triplicates. Data are shown as mean ± SD, *P < 0.05, **P < 0.01, N.S no significance.