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. Author manuscript; available in PMC: 2023 Jan 1.
Published in final edited form as: Pharmacol Res. 2021 Dec 9;175:106030. doi: 10.1016/j.phrs.2021.106030

Fig. 7.

Fig. 7.

TLR4 mutation protected kidney from injury in Ang-II hypertension. (A) Kidney injury molecule-1 (KIM-1) was detected by immunostaining. Representative images from random fields (5 fields/slide/animal). Magnification 60X. Scale bar 40 μM. (B) plasma Lipocalin-2 (LCN2) was measured by ELISA. Data represent mean ± SEM, n=5/saline (control) groups, and n=6/Ang-II treated groups: *P<0.05 vs. TLR4N+saline (control), and †P<0.05 vs. TLR4N+Ang-II. (C) Western blot images of kidney tissue KIM-1 and Lipocalin-2 expression. GAPDH was used as loading control. Bar graphs represent fold change of protein expression. Data presented as mean ± SEM, n=5/saline (control) groups, and n=6/Ang-II treated groups: *P<0.05 vs. TLR4N+saline (control), and †P< 0.05 vs. TLR4N+Ang-II of respective proteins.