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. 2022 Jan 13;13(1):55. doi: 10.1038/s41419-021-04496-9

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — A The colony formation ability of GBM cells was detected by soft agar assay. B Orthotopic implantation experiment was performed to assess the in vivo tumor formation capabilities of ZC3H15-knockdown and control cells. C The survival curve of mice was plotted based on the living days of mice after the in-situ injection. D The expression levels of ZC3H15, CBL, and EGFR were stained and showed in the representative section of tumor samples of GBM patients. ZC3H15-H, ZC3H15 high expression; ZC3H15-L: ZC3H15 low expression. E Model of the impact of the ZC3H15 on regulating cell proliferation, migration, invasion, and tumorigenesis of GBM cells. The data were expressed as mean ± SD. Student’s t test was performed to analyzed significance. *P < 0.05, **P < 0.01, ***P < 0.001.