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. 2021 Jan 26;29(1):101–121. doi: 10.1038/s41417-021-00293-w

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© The Author(s), under exclusive licence to Springer Nature America, Inc. part of Springer Nature 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — A The putative binding site between miR-486-3p and circFLNA was predicted by RNA22. B The result of luciferase reporter assay validated that circFLNA sponged miR-486-3p in SK-MES-1 and A549 cells. C QRT-PCR was used to detect the circFLNA expression in the cancer tissues and paracancer tissues. D Pearson was used to analyze the correlation between circFLNA and miR-486-3p. All the experiments were conducted three times (^**P < 0.01, vs. IC). I miR-486-3p inhibitor, IC inhibitor control.