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. 2022 Jan 3;12:764130. doi: 10.3389/fphar.2021.764130

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Copyright © 2022 Wang, Yang, Chen, Chen, Li, Han, Kamili, Wu and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Rb2 inhibited lipid accumulation in plaque and peritoneal macrophages of mice. (A) Representative image of Oil red O staining in the thoracic aorta from the Ad-NC group, Ad-miR-216a group, Ad-NC + Rb2 group, and Ad-miR-216a + Rb2 group. Scale bar = 200 μM or 50 μM. (B) Quantification of the percentage of lipid (of the total lesion area) in the thoracic aorta from the Ad-NC group, Ad-miR-216a group, Ad-NC + Rb2 group, and Ad-miR-216a + Rb2 group. (C) The mRNA expression of ABCG1 and ABCA1 of peritoneal macrophages from the Ad-NC group, Ad-miR-216a group, Ad-NC + Rb2 group, and Ad-miR-216a + Rb2 group. **p < 0.01, &&p < 0.01, compared to the Ad-NC group. #p < 0.05, compared to the Ad-miR-216a group. n = 5 for each group.