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. 2022 Jan 18;22:77. doi: 10.1186/s12885-022-09180-5

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — High S100A2 expression is correlated with clinicopathologic features in patients with endometrial carcinoma. (a) Differences in S100A2 expression in UCEC tissues and adjacent normal tissues. (b) Differences in S100A2 expression in normal samples from the GTEx combined adjacent UCEC tissues and UCEC samples. (c) Differences in S100A2 expression in UCEC samples and paired adjacent samples. (d) ROC curve for S100A2 in normal samples from the GTEx combined adjacent UCEC tissues and UCEC samples. (e) S100A2 protein levels were markedly upregulated in tumor tissues compared with that in non-paired normal tissues. (f) Representative images of S100A2 expression in endometrial carcinoma tissues and their normal controls