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. 2022 Jan 19;41:28. doi: 10.1186/s13046-021-02211-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — TFEB promotes EC migration depending on ERRα via EMT signaling (A-B) Wound healing of TFEB-ERRα regulation in KLE and ECC-1 cells. (C-D) TFEB regulates ERRα, E-cadherin, and vimentin expression in EC cells is analyzed using RT-qPCR. (E–F) The effect of XCT790 treatment for 24 h on TFEB over-expressing in EC cells. *, p < 0.05. Statistical tests: Student’ s t-test.