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. 2022 Jan 20;22:84. doi: 10.1186/s12885-021-09165-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Correlation analysis between ACK1, immune cells and immunomodulators processed by the TIMER database and the TISIDB website. A Correlation between ACK1 gene copy numbers and immune cell infiltration levels in COAD. *p < 0.05; **p < 0.01; ***p < 0.001. B The correlation between the expression level of the ACK1 gene and different immune cells in COAD by the TIMER database. C Correlation between ACK1/TNK2 gene expression and different immune cell subgroups by the TISIDB website