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. 2022 Jan 20;17:13. doi: 10.1186/s13000-021-01182-4

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — STAT3 is highly expressed in cervical cancer patients and is negatively correlated with LC3B.A. the expressions of STAT3 and LC3B in cervical cancer patients; B. The STAT3 immunoreactive score (IRS) is negatively related to LC3B IRS; C and D. P-STAT3 and LC3B were detected by western blotting, then analyzed by Image J; E. The mRNA expression of LC3B in normal cervical tissue was significantly higher than that in cervical cancer by GDS3233.F. The mRNA expression of STAT3 in normal cervical tissue was no different with that in cervical cancer. G. STAT3 correlates positively with LC3B at the mRNA level. (“ns” no significance, “ * ” p<0.05, “ ** ” p<0.001)