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. 2022 Jan 20;17:13. doi: 10.1186/s13000-021-01182-4

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — STAT3 promotes proliferation, migration and clonal formation of cervical cancer cells. A STAT3 guide-RNA sequence; B The knockout and knockdown efficiency was verified by western blotting; C-E. cck8, clone formation, transwell assays detected the proliferation ability, clone formation and migration ability; F Representative appearance of subcutaneous tumors, the tumor weight and the tumor growth curve in the study; G The expression of STAT3 in tumor tissues was detected by western blotting. (“ns” no significance, “ * ” p<0.05, “ ** ” p<0.01, “ *** ” p<0.001, “ **** ” p<0.0001)