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. 2022 Jan 14;14(2):413. doi: 10.3390/cancers14020413

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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(A). Genomic sites with altered chromatin accessibility (THSSs) in vascular malformations of mice injected with MS1 cells expressing GNAQ-Q209L (GNAQ-Q209L) or not (CTL). (B). Distribution of differential ATAC-seq peaks with respect to gene features. (C). Gene ontology biological process enrichment analysis of genes containing differential ATAC-seq peaks. Asterisk denotes pathways involved in angiogenesis or vasculogenesis. (D). List of genes with differential chromatin accessibility; Asterisk denotes genes involved in angiogenesis or vasculogenesis. (E). Examples of differential ATAC-seq peaks in two genes related to angiogenesis.